STAT3 Inactivation and Induction of Apoptosis Associate With Fluoxetine-inhibited Epithelial-mesenchymal Transition and Growth of Triple-negative Breast Cancer In Vivo
OncologiaSobre o estudo
- Desenho
- Metanálise
- Nível de evidência
- 1 · Evidência consolidada
- Revista
- Anticancer research
- Publicado em
- 1 ago 2022
- Citações
- 16
O resumo deste estudo é do publisher e não tem licença aberta: por isso mostramos aqui só os dados do estudo, e o texto fica na fonte original, nos links abaixo.
O estudo original
STAT3 Inactivation and Induction of Apoptosis Associate With Fluoxetine-inhibited Epithelial-mesenchymal Transition and Growth of Triple-negative Breast Cancer In Vivo
Pen-An Liao, Pei-Yi Chu, Zhao-Lin Tan, Fei-Ting Hsu, Yang-Cheng Lee, Hsing-Ju Wu
Descritores MeSH
- Triple Negative Breast Neoplasms
- Cell Movement
- Female
- Fluoxetine
- Humans
- Gene Expression Regulation, Neoplastic
- Animals
- Cell Line, Tumor
- Cell Proliferation
- STAT3 Transcription Factor
Veja também
Estudos relacionados
- Intermittent Fasting Attenuates Obesity-Induced Triple-Negative Breast Cancer Progression by Disrupting Cell Cycle, Epithelial-Mesenchymal Transition, Immune Contexture, and Proinflammatory Signature
- A Cell-Based Systematic Review on the Role of Annexin A1 in Triple-Negative Breast Cancers
- LINC00657 exhibits oncogenic properties in prostate cancer and may serve as a prognostic biomarker in cancer
- Cold atmospheric plasma-activated medium for potential ovarian cancer therapy